
CENTER FOR THE PROFESSIONAL STUDY OF
ATTENTION DEFICIT HYPERACTIVITY DISORDER (AD/HD)
Dr. Robert R. Erk, Ed.D., is the Director of the Center for the Professional
Study of Attention Deficit Hyperactivity Disorder. Dr. Erk is a Professor of
Counselor Education and Educational Psychology in the Department of Educational
Studies at the University of Tennessee at Martin, TN 38238. Dr. Erk teaches
graduate coursework in school and mental health counseling, served as a public
school counselor, founded the Professional Interest Network for Children With
AD/HD for the American School Counselor Association, is a Licensed Professional
Counselor and Marital and Family Therapist in Tennessee, and is the parent of a
son diagnosed with AD/HD. Dr. Erk is a past president of the Southern
Association for Counselor Education and Supervision.
The Center strives to provide resources or information on AD/HD for parents,
counselors, and teachers, conducts inservices and workshops on AD/HD at the
state, regional, and national levels, and maintains a research and publication
agenda on the disorder. This content of this webpage is designed to be readable
for parents, teachers, and counselors.
A SYSTEMS ORIENTED APPROACH TO AD/HD:
FAMILIES, SCHOOLS, AND COMMUNITIES WORKING TOGETHER
The Center is dedicated to advocating and promoting a Systems Oriented
Approach To AD/HD: Families, Schools, and Communities Working Together. This
approach is centered on the development of the child with AD/HD and their
ability to function more effectively in the home, in the school, and in the
community. The systems oriented approach focuses on the education and training
on AD/HD that has been conducted or occurred in the major systems that are
connected to the affected child's life. The major systems considered here are
the following:
THE FAMILY SYSTEM. For example, the parents and siblings
comprise the family system; the family system may also be composed of extended
or blended family members. It is important for parents (or all family members)
to increase their knowledge and understanding of the AD/HD, realize that the
disorder has often brought about high stress levels for its members, and that
the problem behaviors can be managed more effectively. Importantly, the family
needs to understand that the unity of the parents and siblings in fully
accepting the child with AD/HD and working collaboratively to foster a
supportive and healthy family environment is crucial. This includes the
parents' taking responsibility for seeking and supporting appropriate treatment
interventions or regimens for the AD/HD.
THE SCHOOL SYSTEM. For example, the school officials,
teachers, classmates, and peers comprise the school system. In many instances,
by not being educated or trained on the symptoms and problematic behaviors that
are often connected to the disorder, school personnel or educators may
inadvertently exacerbate the symptoms or problematic behaviors of the child
with AD/HD. Unfortunately, classmates or peers at school often relentlessly
tease, taunt, devalue, and socially shun many children with AD/HD. For children
with AD/HD, these social-emotional experiences with classmates or peers can be
indelible in theirmemories. Therefore, schools are the place of choice for also
educating students about the AD/HD symptoms and the problem behaviors they may
observe.
THE SCHOOL TO FAMILY SYSTEM. For example, the school
personnel or educators, parents, and siblings comprise the school to family
system. The school and family system need to become clinical allies or form a
therapeutic alliance for the identification and behavioral management of the
child with AD/HD. Moreover, school systems may be subject to complying with the
provisions of the federal acts such as the Individuals With Disabilities
Education Act (IDEA), Section 504 of the Vocational Rehabilitation Act, and the
Americans With Disabilities Act (ADA). These federal acts can apply to students
diagnosed with AD/HD from elementary school through the college years. Schools
are becoming increasingly aware of their legal obligation to appropriately
serve children and adolescents with AD/HD. For example, these federal acts have
often served as an impetus and pointed out the need to increase the inservice
education and training for educators on AD/HD children. School systems may need
to increase their vigilance for identifying and supplying appropriate
interventions, accommodations or variations in classroom instruction and testing
practices for AD/HD children. At school, the counselor may consent to serve as
a resource person or a case manager for appropriate services or treatment for
the child with AD/HD. By forming a school to home partnership and coordinating
together the interventions or treatment plan, the likelihood that the AD/HD
child can perform more satisfactorily is optimized.
THE FAMILY TO COMMUNITY SYSTEM. For example, the parents
and siblings along with the individuals in their community comprise the family
to community system. Locale and its prevailing attitudes and sentiments are
often the principal components of the community system. However, for the family
with an AD/HD child, the community system can refer moreappropriately to the
development of social interaction, social cohesion, moral support, and contains
the potential for fostering a therapeutic resource or alliance. For example, it
is crucial that our communities become better informed or educated on children
with AD/HD and their problematic behaviors, rather than focusing on and
labeling the dysfunctional elements or parts of the child with AD/HD. The
family to community system is the place where the family lives, attends school,
engages in recreational activities, has intimate ties, and it is the community
that can act as a facilitator for fostering the development of positive mental
health for many children with AD/HD. Finally, it is the community that can
strongly affect the course of events or the outcomes for families with an AD/HD
child.
CHANGING AND RECONSTITUTING SYSTEMS
From a systems oriented approach to AD/HD, all of these systems can be
perceived of as changing and reconstituting themselves by becoming more open to
scientific, medical, and behavioral explanations for the disorder. For example,
many persons who come into contact with an AD/HD child do not know that the
child's behavior is not willfully or deliberately planned to confront,
undermine, or provoke their parents, teachers, or peers. Instead, becoming
educated and understanding how this complex disorder can have life-long effects
(e. g., academic, social-emotional, occupational) on the child with AD/HD, and
that the actions taken (e. g., positive or negative) in any one system can have
reciprocal or circular effects. For example, the AD/HD child's problematic
behaviors or actions in any one system can in-turn influence the family,
school, and community's reactions to the child. It should be remembered that
these reciprocal actions and their consequences can often be imprinted into the
life of the child or adolescent with AD/HD.
CLOSING REMARKS
These systems can be viewed as forces working for either the good or to the
detriment of the child with AD/HD. There is an interconnectedness among the
systems, the actions in one system or any part of it can affect the health or
dysfunctionality of the child. The child with AD/HD is an interacting and
interactive member of these systems. They are powerful parts of the child's
life and they frequently exert a strong influence over not only the child's
behaviors, but certainly how their feelings, values, and attitudes evolve
towards themselves and others.
A note of caution is probably in order, change in how these systems view and
interact with AD/HD children and adolescents will likely be gradual or long-term.
By necessity there will need to be a heavy investment in education and training
on the disorder.
MULTIDIMENSIONAL TREATMENT FOR AD/HD
Through a systems oriented approach to AD/HD and a multidimensional
treatment plan the development of children and adolescents with AD/HD can be
enhanced. Perhaps the most important point of information to emerge from the
last several years of research on AD/HD is that no single intervention alone is
probably going to be successful. The multidimensional treatment approach,
moreover, needs to be long-term so as to optimize the child's development. The
goal of multidimensional treatment should not be to find a cure. The goal is to
provide interventions or strategies that can be important for managing this
often chronic and disabling condition. The multidimensional treatment plan
enables the professionals and parents to attack the AD/HD from as many avenues
as are feasible. Interventions or treatment typically includes some combination
of the following:
PARENT EDUCATION. Education for parents and siblings on the
AD/HD symptoms and problem behaviors that are associated with the disorder is
the necessary first-step. Parent education can be the benchmark upon which
further progress for the family may be measured. For example, parent education
can explain how the AD/HD symptoms can frequently create in the home and at
school, the myriad of problematic behaviors that are often considered
disruptive or maladaptive to parents and teachers. Parent education can
normally be accomplished in four to six well-planned one-hour sessions from a
professional (or a parent support group) that is well-informed and experienced
with the disorder. From parent education on AD/HD, parents can be relieved of
the guilt that they were somehow the cause of the disorder, they can acquire a
better understanding of the disorder, and they can focus their efforts towards
improving the quality of life for their AD/HD child.
TEACHER EDUCATION. Educating teachers and school officials
on the academic, behavioral, and personal-social problems that students with
AD/HD often exhibit provides them the information that they need to work more
effectively with these children. As teachers and school officials become better
educated about AD/HD students, they can work collaboratively to maximize the
interventions or strategies that are targeted at improving the academic,
behavioral, and social functioning of these children. Through teacher education
on the AD/HD, issues such as educational negligence or malpractice with AD/HD
children can then be taken out of the equation. Increasingly, many state
departments of education are urging local educational agencies or school
districts to come into compliance with federal acts or statutes that can
address the need for the identification and treatment of children with AD/HD.
INDIVIDUAL COUNSELING. Professional counselors who work
with AD/HD children and adolescents should acknowledge at the onset that many
of these children can be a formidable challenge. Often children with AD/HD tend
to be in a state of denial about their disorder and their problems. Moreover,
many of these children can be oppositional in their attitudes, distrustful of
authority figures, have low self-esteem, and see most of their difficulties or
problems as being caused by others such as teachers, parents, and peers.
Counselors who are remarkably patient and understanding, who can endure more
relapses than progress in the beginning sessions or phases of counseling, and
who can remain a positive force in the lives of these children often have the
most success in forging a therapeutic relationship. Professional counselors
should be aware that many children with AD/HD can have coexisting conditions
such as learning disabilities, oppositional defiant behavior, excessive
anxiety, language usage problems, anger control problems, and can experience
depression.
BEHAVIORAL MANAGEMENT TECHNIQUES. Educators and parents
should consider that behavioral management techniques or strategies such as
time-out, negative reinforcement, cost response measures for inappropriate
behavior, contracting, earning rewards and special privileges for appropriate
behavior, and cognitive behavioral training (e. g., self-instructional
training) coupled with appropriate medical treatment seems to be the most
effective treatment for many children with AD/HD. In collaboration with the
parents and the school, professionals, parents, and teachers can develop
priorities for employing behavioral management techniques that target or
address the child's problematic behaviors at school and in the home. It needs
to be remembered that behavioral techniques or strategies often need to be
varied, adjusted, or changed to meet the problematic behaviors or situations
that AD/HD children typically exhibit. It should also be remembered that there
is a neurobiological basis for the AD/HD. This suggests that AD/HD children
will often need more behavioral structure, more time to come into compliance,
and more varied or unique reward systems to effectively promote change in their
lives. Furthermore, in June, 1997 changes in IDEA were signed intolaw by
President Clinton. One of the key changes, for example, that could be pertinent
to schools and parents is the provision dealing with discipline. It is not
usual for a child with AD/HD to become involved at some point in their school
career, in a disciplinary process thatinvolves their behavior. In determining
whether suspension or expulsion from school is allowable, the school or
committee must consider: (a) the child's ability to understand or comprehend
accurately what he or she did, and (b) their ability to control the behavior
involved in the event or problem. The control part of this change can have
important factorssuch as did the child have the opportunity to receive appropriate
medication and interventions or treatment for the disorder.
SELF-ESTEEM EDUCATION. The major goal of parents, teachers,
and counselors for many children with AD/HD needs to be the restoration of
their self-esteem. Without realizing it, many parents, teachers, and peers have
often been excessively critical of the child, repeatedly complained about their
misbehaviors, and unfortunately demeaned or belittled many of their efforts or
struggles to improve themselves. For persons who come into contact with AD/HD
children, it should be remembered that they have endured many more frustrations
and failure experiences in their young lives than other children. At every
opportunity, parents and teachers should seek to create or take advantage of
opportunities that can foster self-esteem and accomplishment in AD/HD children.
SOCIAL SKILLS EDUCATION. There usually needs to be a strong
instructional and training program on social skills education. Unfortunately
due to their disorder, many AD/HD children often have a limited or inadequate
repertoire of social skills available to them. From the standpoint of many
teachers and peers, poor or deficient personal-social skills stand-out almost
immediately as the area most in need of improvement. For example, almost everything
the child with AD/HD does is viewed as socially different or inappropriate,
they often feel that they are being excluded from active participation in their
classrooms, and they often feel that their peers further exclude them from many
social activities or games at recess. In many instances, the child with AD/HD
can also be disinvited by classmates or peers from attempting to play with them
after school or on weekends. Instruction and real-life practice in acquiring an
improved repertoire of social skills across settings (e. g., school, home)
often needs to be a priority for many AD/HD children.
FAMILY COUNSELING. Family counseling is frequently
recommended because the AD/HD typically disables the family's communication
patterns, is a root cause of family disagreements or arguments, and can lead to
marital dysfunction, sibling dissension, and the social shunning of the family
by others in their community. Many families need to come to the realization
that the child cannot be separated from their disorder. Accepting and nurturing
the child with AD/HD from childhood, to adolescence, to adulthood despite the
multiple frustrations can be the best form of therapy. For children with AD/HD,
the unity of the parents can be crucial to the future direction of their young
lives.
In summary, a systems oriented approach to AD/HD and multidimensional
treatment for the disorder can compliment and reinforce each other. They share
the priority of providing and sharing education on the disorder and fostering a
safer psychological environment for providing treatment or services for AD/HD
children. It is within the family, school, and community systems that children
with AD/HD can learn how to live more successfully and have more fulfilling
lives.
THE COLLABORATIVE MULTIMODAL TREATMENT STUDY OF CHILDREN WITH ATTENTION DEFICIT/HYPERACTIVITY DISORDER (AD/HD)(MTA STUDY)
The MTA Study constitutes a landmark in the history of treatment research in
child psychopathology; it is the largest single study of its kind ever undertaken
for one disorder (AD/HD)(Barkley, 2000). Because the answers to such questions
can only be pursued through large scale efforts that generate sufficient sample
sizes for powerful tests of treatment effectiveness (e. g., so treatment
outcomes can be more properly compared and evaluated), the National Institute
of Mental Health (NIMH) launched a collaborative multisite (N=6) clinical trial
of Multimodal Treatment of children diagnosed with AD/HD. The NIMH-MTA Study
has been in the field since 1992; between 1992 and 1994, 579 children (aged 7-9
years, 11 months) were recruited at participating sites in the United States
and one in Canada (Montreal). All the children had primary diagnoses of AD/HD,
Combined Type, using Diagnostic and Statistical Manual for Mental Disorders
(DSM-IV; American Psychiatric Association, 1994) criteria. In addition to the
AD/HD, comorbid or co-occurring aggressive spectrum disorders (e. g.,
oppositional defiant disorder [ODD], conduct disorder [CD]), anxiety disorders,
and affective disorders occurred in the MTA sample at these respective
prevalence rates 54%, 34%, and 4%. The subjects in the sample were 61%
Caucasian, 20% African-Americans, and 8% Hispanic; males constituted 80% and
females 20% of the sample. Children were randomly assigned to one of four
experimental groups: (1) an intensive, multifaceted behavior therapy program
(BEH) comprised of three integrated components (Parent Training, School
Intervention, Child Treatment anchored in a Summer Treatment Program): this
program can be referred to as "psychosocial treatment" because of its
largely behavioral procedures; (2) a carefully dose-adjusted and monitored
medication management program consisting of methylphenidate and other
stimulants (MED-MGT); (3) a careful and well integrated combination of the
previous two (COMB); and (4) a community comparison group (CC). The CC
comparison group allowed for a comparison of the three intensive manual-based
treatments to the treatment delivered in the community (e. g., local community care
resources). Treatment occurred over 14 months, and assessments were taken at
baselines of 3, 9, and 14 months. After this comprehensive assessment, the
global effects of the treatment for the MTA Study of BEH, MED, COMB, and CC
groups are summarized below. It should be kept in mind that the MTA Study was
indeed large; many of the variables contained in the study are yet to be fully
scrutinized for their specific roles. Nonetheless, here are some of the
essential findings gleaned from the study.
First, there were generally superior effects for COMB and MED-MGT compared
to BEH or CC on the primary characteristics of AD/HD, including some comorbid
conditions. This should not be unexpected; an array of interventions or a
multidimensional approach to treatment has long been advocated in the
management of this disorder (Barkley, 1990, 1995; Erk, 1995, 1997, 2000).
Second, the BEH or psychosocial treatment approach "alone" did
produce some effects despite being limited. The amount of gain here probably
should have occurred because the study seemed heavily weighted toward the BEH
or psychosocial treatment approach. However, Barkley (2000) questioned if such
an extensive BEH program could be justified (e. g., now or ever) in homes and
schools (e. g., due to its extensive scale). Relevant to the gains made in this
approach, Barkley (2000) speculated that the modest differences found between
the BEH and CC groups in the MTA Study might be more due to information and
therapist attention; these could have accounted for the small differences (e.
g., in favor of the BEH group). For example, numerous parent education and
behavioral training programs for parents with children diagnosed with AD/HD are
already in existence and might obtain much the same results when properly
employed. The BEH approach to treatment "means" parents are willingly
becoming involved in parent education and training on the AD/HD, and they are
learning and actively practicing a range of behavioral interventions designed
to benefit the family and the child.
Third, the MED component, which entails no skill training for parents or
teachers, achieved equal or superior effects to BEH across nearly all of the
outcome domains (e. g., parent-child relations, familial and school
functioning) examined in the study (Barkley, 2000). The MTA Study, coupled with
the incoming lines of scientific and medical information (e. g., PET scans,
Human Genome Project), should further serve to prompt parents and practitioners
to be increasingly receptive to MED-MGT as one of the key players for obtaining
improved therapeutic results (e. g., especially where the AD/HD is moderate to
severe).
Fourth, only in COMB treatment, not in BEH alone, were there the greatest
reductions in negative-ineffective discipline. This finding should be
considered among the most vital when treatment options are being planned. For
example, success for COMB treatment was evident for important school-related
outcomes (e. g., reduction in teacher reported disruptive behavior). Moreover,
this finding would appear to be related to reductions in negative and
ineffective parenting practices in the home (e. g., reciprocal or parent-child
interactions; Erk, 1997). Therefore, the home and school environments benefited
reciprocally from the COMB treatment.
Fifth, when comorbid symptomatology (e. g., ODD, CD) and functional skills
(e. g., academic and social skills) were considered, the group differences
among all the treatment groups were smaller. For example, only the COMB
treatment was consistently superior to CC when the comorbid conditions were
identified. It should be remembered that comorbid symptomatology can be a major
disrupter to any treatment that is being utilized; what this finding from the
study reinforces is the need for careful assessment of comorbid conditions and
that these conditions deserve as much attention (e. g., treatment) as the AD/HD
itself. Unfortunately for the child, if the comorbid conditions remain
undiagnosed and untreated, the comorbid conditions will often be mistakenly identified
as the primary problems. This can lead to the AD/HD remaining at the
"core" of the person's difficulties while the disorder can continue
as undiagnosed and untreated (Erk, 1997, 2000).
Sixth, COMB significantly outperformed MED-MGT with a small-to-moderate
effect size (Hinshaw et al., 2000). This should not be entirely a surprising
result; MED-MGT alone is not considered a prudent approach when it is to be
used in isolation. Barkley (1990, 1995) recommended that parents should not
invest in a MED-MGT only treatment plan. This is because with a MED-MGT only
plan, the child is obviously deprived of opportunities to learn or acquire an
improved behavioral repertoire (e. g., social and academic skills, enhanced and
self-esteem).
Seventh, and last, although overall findings were consistent for boys versus
girls, for children with and without prior medication treatment, and subjects
with and without disruptive comorbidity, two baseline variables (e. g.,
comorbidity with anxiety disorder and socioeconomic status) had the effect of
moderating some treatment outcomes. A major limitation of the MTA Study may be
the exclusive use of the AD/HD Combined Subtype; it was presumed that
hyperactivity, impulsivity and inattention components are incorporated into the
Combined Subtype. However, for example, in the Inattentive Subtype there may be
different findings and implications for treatment identified for children who
are diagnosed with this specific subtype (e. g., see Erk, 2000). Future studies
will need to address issues such as this one (e. g., specific subtypes) and the
comorbidities that most often co-occur. The MTA Study and its employment of a
multimodal treatment model paves the way for further research and treatment
appropriate for children with AD/HD. For example, earlier clinical research (e.
g., using parent behavioral training with medication management) for AD/HD
children has yielded positive results; this is reinforced by findings contained
in the MTA Study (Barkley, 2000). For professionals and practitioners, there is
certainly much that is positive and informative from the MTA Study.
Undoubtedly, more clinical results will emanate from this research for years to
come; the MTA Study should be clinically, scientifically, and educationally
examined for every drop of information it can yield on AD/HD and its
management. The MTA Study was mammoth and unparalleled in the field of child
treatment; for this reason it can be expected to take years, if not decades, to
tease out all data that is has to offer researchers and professional
practitioners (e. g., teachers, school and mental health counselors,
psychologists, physicians).
REFERENCES/SUGGESTED READINGS ON AD/HD
American Psychiatric Association (1994). Diagnostic and Statistical Manual of Mental Disorders (4th ed.), Washington, DC: Author.
Barkley, R. A. (1990). Attention Deficit Hyperactivity Disorder: A
Handbook for Diagnosis and Treatment. New
York: Guilford Press.
Barkley, R. A. (1995). Taking Charge of AD/HD: The Complete Authoritative
Guide for Parents. New York: Guilford
Press.
Barkley, R. A. (2000). Commentary on the multimodal treatment study of
children with AD/HD. Journal of Abnormal Child Psychology, 28, 595-598.
Copeland, E. D., & Love, V. L. (1991). Attention, Please!: A
Comprehensive Guide to Parenting Children With Attention Disorders and
Hyperactivity. Atlanta, GA: Southeastern
Psychological Institute Press.
Erk, R. R. (1995). The Conundrum of Attention Deficit Disorder. Journal
of Mental Health Counseling, 17, 131-145.
Erk, R. R. (1995). A Diagnosis of Attention Deficit Disorder: What Does It
Mean For School Counselors? The School Counselor , 42, 292-299.
Erk, R. R. (1995). The Evolution of Attention Deficit Terminology. Elementary
School Guidance and Counseling, 29, 243-248.
Erk, R. R. (1997). Multidimensional Treatment of Attention Deficit Disorder:
A Family Oriented Approach, Journal of Mental Health Counseling, 19,
3-22.
Erk, R. R. (1999). Attention deficit hyperactivity disorders: Counselors,
laws, and implications for practice. Professional School Counseling, 2, 318-326.
Erk, R. R. (2000). Five frameworks for increasing understanding and
effective treatment of attention-deficit/hyperactivity disorder: Predominantly
inattentive type. Journal of Counseling and Development, 78, 389-399.
Erk, R. R. (2004). Counseling treatment for children and adolescents with
DSM-IV-TR disorders. Upper Saddle River,
NJ: Merrill Prentice Hall.
Friedman, R. J., & Doyal, G. T. (1992). Management of Children and
Adolescents With Attention Deficit Hyperactivity Disorder. Austin, TX: Pro-Ed.
Hallowell, E. M., & Ratey, J. J. (1994). Driven to Distraction: Recognizing and Coping With Attention
Deficit Disorder from Childhood through Adulthood. New York: Touchstone.
Hinshaw, S. P. et al., (2000). Family processes and treatment outcome in the
MTA: Negative/ineffective parenting practices in relation to multimodal
treatment. Journal of Abnormal Child Psychology, 28, 555-564.
Hosie, T. W., & Erk, R. R. (1993, January). American Counseling
Association Reading Program: Attention Deficit Disorder. American Counseling
Association Guidepost, 35, 15-18.
Learner, J. W., Lowenthal, B. & Learner, S. R. (1995). Attention
Deficit Disorders: Assessment and Teaching.
Pacific Grove, CA: Brooks/Cole.
Nadeau, K. G. (1995). A Comprehensive Guide to Attention Deficit Disorder
in Adults: Research, Diagnosis, and Treatment.
New York: Brunner/Mazel.
Reif, S. F. (1993). How To Reach And Teach ADD Children. West Nyack, NY: Center for Applied Research In
Education
Shaywitz, S. E., & Shaywitz, B. A. (1992). Attention Deficit Disorder
Comes of Age: Toward the Twenty-First Century.
Austin, TX: Pro-Ed.
HEALTH-RELATED
QUALITY OF LIFE
IN CHILDREN, ADOLESCENTS, AND YOUNG ADULTS DIAGNOSED WITH
ATTENITION-DEFICIT/HYPERACTIVITY DISORDER
Attention-Deficit/ Hyperactivity Disorder (AD/HD) significantly affects the health-related
quality of life for children, adolescents, and adults. There is a
substantially lower health-related quality of life across multiple
domains (e.g., physical, psychosocial) when compared with normative data for
individuals without AD/HD (Klassen et al., 2004). Physical domains can include:
physical health and functioning (e.g., including role and/or social limitations
as a result of physical health). Psychosocial domains can include:
social-emotional-behavioral problems, mental health, self-esteem, self-neglect,
and impact on family functioning.
Klassen et al. (2004) found that the health-related quality of life
was correlated with two factors: (1) AD/HD symptom severity, and (2) the
presence of comorbid or co-occurring psychiatric disorders. Research has
established that comorbidity or co-occurring disorders are common in cases of
AD/HD (Barkley, 1998; Pliszka et al., 1999). Simply put, the severity of the
AD/HD and the co-occurring disorders are an important predictor of the
health-related quality of life for many of these individuals. The toll
taken due to the AD/HD and co-occurring disorders on his/her health and/or
safety cannot be emphasized strongly enough.
AD/HD: A
LIFESPAN DISORDER
Although AD/HD has historically been categorized as a childhood disorder, it
is now regarded as a lifespan condition (e.g., into the adult years) (Barkley,
1998; Resnick, 2000). Follow-up studies of AD/HD children into adolescence and
the adult years indicate the disorder frequently persists and it is associated
with significant psychopathology and dysfunction (e.g., social, emotional,
occupational) in later life (Wilens et al., 2002; Weiss & Murray, 2003).
Moreover, follow-up clinical studies provide compelling evidence for the
continuation of AD/HD into adulthood, though the rate at which this occurs
remains unclear. For example, prior studies showed the persistence of AD/HD
symptoms into adolescence (e.g., 50%-75%) and adulthood (e.g., 4%-60%)
(Hechtman, 1992; Mannuzza et al., 1993). However, more recent clinical studies
based on the DSM-III-R (when compared to previous research) have indicated
higher persistence rates of 75% for AD/HD into young adulthood (Biederman et
al., 1993).
Barkley (2003) emphasized this point: Simply because the severity levels of
symptoms might decline over development, this did not mean that children and
adolescents with AD/HD are necessarily out growing their disorder relative to
normal children. In many cases of AD/HD, the disorder is associated with
profound impairment in the adult years (Weiss & Murray, 2003). While the
AD/HD remains at the core of their lives, the assessment and treatment of
co-occurring disorders cannot be left unattended (Erk, 2004a).
The differential diagnosis of comorbid conditions can be difficult, but it
is necessary because individuals with AD/HD have high rates of oppositional,
conduct, mood, anxiety, learning, and personality disorders, substance use and
abuse, and increased risk of suicide (e.g., at a higher rate than found in the
general population) (Barkley, 1998, 2003; Biederman et al., 1991,1993; Brown,
2000; James et al. 2004).
AD/HD AND
NEUROBIOLOGY
Theories and research on AD/HD have been constructed along both neurobiological
and neurochemical lines (see Barkley, 1997, 1998, 2003; Castellanos et
al., 1994; 1996; Erk, 2000, 2004b). Barkley (1997) believed a “lack of
behavioral inhibition” in individuals with AD/HD is at the core of the
disorder. For example, behavioral inhibition and/or executive brain functions
are impaired and cannot come online to work effectively (e.g., to serve the
best interests of the individual). In other words, there are executive and
inhibitory brain deficits that are affiliated with the AD/HD itself (as cited
in Barkley, 2003). In essence, this is due to the neurobiology and
neurochemistry of the brain. There is sufficient neuroscientific data to
document that AD/HD is the result of neuroanatomical and neurochemical
abnormalities that reside in the brain (Barkley, 1997, 2003; Lavenstein, 1995;
Quinn, 1995). Fundamentally, AD/HD can be conceived of as a neurobiological or
neurobehavioral disorder.
A biological explanation is not inclusive and there is not a biological
destiny for persons with AD/HD. There is an important role for environment
and/or learning in the lives of all individuals (Gredler, 2005). However, there
remains an “inseparability” of the brain, thinking, and behavior, Therefore, a
parent, teacher, clinician or counselor, cannot assess and/or understand how
“problem areas” might have developed without a basic grounding in how these
problems originated, evolved, and progressed (for more information on AD/HD and
neurobiology see Barkley, 1997, 1998, 2003; Castellanos et al., 1994, 1996;
Erk, 2000; 2004a; Nadeau, 1995; Quay, 1997; Zemetkin et al., 1986,1990,1996).
Mohr (1995) emphasized that client deterioration tends to be more prevalent
when disorders or psychopathologies in the client are misunderstood,
under-evaluated or under-estimated. Enlarging the understanding or
comprehension of the cognitive, emotional, and behavioral dysfunctions of
individuals with AD/HD; particularly, how they have come into being is vital to
an enhanced perspective for treatment planning (Erk, 2000; 2004a).
AD/HD AND COMORBID DISORDERS:
A POTENTIALLY
LIFE-THREATENING MIXTURE
The impact that mixtures of AD/HD and co-occurring disorders have on the
lives of these individuals can be described as potentially “life-threatening.”
Life-threatening, in the sense, the lives of these individuals contain
increased “risk factors.” It is clear that the impact of AD/HD and co-occurring
disorders need to be evaluated on a case-by-case basis. However, individuals
who have severe AD/HD symptoms and comorbid disorders, impairment and/or
dysfunction across domains, psychopathologies in their parents and/or family,
and a dysfunctional family structure are likely to be on a trajectory that can
threaten his/her health and/or safety.
Therefore, it is crucial to ascertain the “current state” and/or “level of
functioning” of these individuals. It is often not satisfactory to take the
client’s word (e.g., client and/or family deceit is not uncommon). For example,
there might be families where the diagnosis of AD/HD was made in childhood
and/or adolescence and no medication or behavioral treatment was made available
(e.g., often allowing the AD/HD and comorbid conditions to advance).
Thus, a “fact-finding” process that encompasses every family member and/or
relative as well as teachers, friends and/or peers must be undertaken. Think of
the fact-finding as the “required preparation” for the intervention or
treatment. Clearly, assessing and knowing about comorbidity in young people is
critical and will impact treatment in a number of ways, including the need to
intervene and address the “multiple problems” presented (Field & Seligman,
2004).
It is beyond the scope of this web page to include all the co-occurring
disorders that might be coexisting with the AD/HD (for more information on
these see Biederman et al., 1991; Pliszka et al., 1999; Barkley, 1998, 2003;
Erk, 2004a).
The two co-occurring disorders that are the focus of this webpage are: Mood
Disorders/Depression and Substance-Related Disorders. There is a concluding
section on suicidal behavior since more that 90% of people who kill themselves
have a diagnosable mental disorder, commonly a Depressive Disorder and/or a
Substance Abuse Disorder (Conwell et. al., 1995).
MOOD
DISORDERS/DEPRESSION
Mood Disorders encompass Major Depressive Disorder, Dysthymic Disorder, and
Bipolar Disorder (Diagnostic and Statistical Manual of Mental Disorders,
fourth edition, text revision [DSM-IV-TR] American Psychiatric Association
[APA], 2000). Bipolar is included because people with this illness have
depressive episodes as well as manic episodes (for more information on Bipolar
Disorder see DSM-IV-TR, Geller & Luby, 1997; Field & Seligman,
2004). The diagnostic criteria listed in the DSM-IV-TR for these Mood
Disorders must be met for a diagnosis.
In general, the presence of a Mood Disorder/ Depression in children and
adolescents was found to significantly increase the likelihood of another
mental disorder also being present. Conduct Disorder, Oppositional Defiant
Disorder, Anxiety Disorders, Learning Disorders, and AD/HD are particularly
common “co-occurring disorders” (Barkley, 1998; Field & Seligman, 2004).
Lewinsohn et al. (1995, 1998) found significant consequences to comorbidity of
Mood Disorders including an increase in suicide, a negative impact on academic
performance, impaired role functioning, and increased conflict with
parents/family.
The co-occurrence of Major Depressive Disorder and a Substance Use Disorder
appears to be the most destructive combination for individuals with AD/HD. Depression
and alcohol abuse, in particular, both increase considerably during adolescence
(Johnson et al., 1993; Lewinsohn et al., 1991). Coexisting depression and
alcohol or substance abuse might be particularly problematic, particularly for
some adolescents or young adults who use a variety of substances to
self-medicate.
Stowell and Estroff (1992) in their study of substance-abusing adolescents
found that 50% had a Major Depressive Disorder and Dysthymic Disorder. King et
al. (1996) explored the predicators of co-occurring alcohol and/or substance
abuse and depression in 103 adolescents. These researchers found that the
depressed substance-abusing adolescents experienced longer and more often
persistent episodes of depression than depressed adolescents who were not
substance abusers. Moreover, depressed adolescents who abused alcohol or drugs
reported more overall behavior impairment than those who were not substance
abusers.
Kovacs et al. (1993) found people with a history of Mood Disorders are five
times as likely to make a suicide attempt during their lives compared to people
with a history of other mental diagnoses. Schaffer et al. (1996) found that 61%
of children and adolescents who committed suicide met the criteria for Mood
Disorder, and of that 61%, 52% met the criteria for Major Depressive Disorder,
22% for Dysthymic Disorder, 10% for Adjustment Disorder with Depressed Mood, 4%
for Bipolar Disorder, and 12% for Depression, Not Otherwise Specified.
Because Mood Disorders and/or Depression are found to have a tendency to
“follow” rather than precede the onset of a co-occurring disorder, identifying
and treating other mental disorders (e.g., that preceded the Mood Disorder
and/or Depression) might prevent, delay, or reduce the impact of depressive symptoms
(Lewinsohn et al., 1998). Parents, clinicians, and counselors should keep in
mind that young people with comorbid disorders are at an increased risk for
further episodes of depression and manifest a greater number of social-skills
and problem-solving deficits (Reinecke, 1995). When alcohol or drugs are
present with these factors, there is added risk for suicide (Field &
Seligman, 2004).
SUBSTANCE-RELATED
DISORDERS
The causes or origins of
Substance-Related Disorders are multifactorial (e.g., based on interactions of
biological, environmental, and personality factors). Researchers have long
noted the “biological” or “family” transmission of substance use disorders,
especially alcoholism (Piazza, 2004). Children and adolescents with AD/HD are
at increased risk for developing alcoholism and other drug addictions,
especially if alcoholism exists in other family members (Wilens, 1998). The
odds of this increased risk for alcohol dependence was 45% greater among
individuals with problem-drinking second- or third-degree relative only, 86%
greater among individuals with a problem-drinking first-degree relative only,
and 167% greater among individuals with at least one problem drinking
first-degree relative and at least one problem-drinking second- or third-degree
relative (Department of Health and Human Services [DHHS], 1997).
Overall, inherited individual and/or biological
differences in the brain are believed to regulate the individual’s sensitivity
to the effects of certain substances and place them at “increased risk” of
compulsive substance use (Piazza, 2004).
“Environmental” risk factors (e.g., discordant parent-child interactions,
unfavorable family structure and function, problems at school and/or in the
community, and economic adversity) cannot be discounted for their potential
roles in the development of alcoholism and other drug disorders. Wilens (1998)
believed that AD/HD-related alcohol and other drug abuse can develop early in
youth (e.g., mid- adolescence) as an attempt to alleviate symptoms of mental
distress associated with chronic failure, feelings of inadequacy, and conflict
with parents and peers.
The “personality structure” is genetically and environmentally determined;
it is built on and modified by environmental influences or experiences (Harris,
1995). The person’s personality cannot be separated from its genetically
embedded components. It should be remembered that the AD/HD would be carried
along with the personality into the environment (e.g., school, community).
Affecting, for example, the way that others will react to the individual with
the disorder (Erk, 2000). Werry (1995) believed that AD/HD should be considered
characteristic of the personality structure of individuals with the disorder.
The “key” here is to understand that the presence of AD/HD is an important
risk factor for the development of Substance Use and Substance-Related
Disorders. For example, disorders associated with alcohol and other drugs tend
to appear earlier and progress more rapidly in persons with AD/HD (Wilens,
1998). Approximately 50% of adults with AD/HD exhibit alcohol and other drug
abuse or dependence (Wilens et al., 1996). Data suggest that the risk of
developing alcohol and other drug disorders at anytime over the lifespan of an
adult with AD/HD is twice that of adults without AD/HD (52% versus 27%
respectively) (Biederman et al., 1995). Elevated rates of alcoholism are
consistently found in the parents of youth with AD/HD (Wilens et al., 1996).
For individuals with AD/HD, who were never treated for the disorder in
childhood or adolescence, substance abuse is a common outcome (Resnick, 2000).
In these particular cases, the potential for alcohol and/or substance addiction
is high. Unfortunately, alcoholics with AD/HD are less likely than those without
AD/HD to remain in alcoholism treatment programs or to achieve moderation or
abstinence (Tarter & Edwards, 1988).
Research supports a significant relationship between AD/HD and alcohol and
other drug disorders, with many symptoms of AD/HD, appearing many years before
the earliest onset of abuse. When alcohol and other drug disorders begin in
adolescence, they run a more severe course than those that appear in adulthood.
Therefore, early treatment strategies should be directed at children and
adolescents with AD/HD before alcohol and other drug use problems develop and
become chronic (Wilens et al., 1997).
KEEP YOUR
EYE ON SUICIDE
In the year 2000, there were approximately 30,000 suicides. Suicide was the
third leading cause of death among 15- to 24-year olds. Suicide was also the
third leading cause of death among children ages 10-14 (National Institute of
Mental Heath [NIMH], 2003). It is the second largest killer of students in the
college population.
Suicide is a tragic; yet, potentially preventable public health problem.
Suicidal behavior is complex. With each case, there are risk factors. Some risk
factors vary with age, gender (e.g., more males are completers, more females
are attempters), and ethic group. Risk factors might include: childhood trauma
(e.g., physical or sexual abuse, domestic violence, disruptions to attachment
figures), mood disorders (e.g., are involved in the development of suicidal
thoughts and behaviors) and addictions (e.g., especially alcoholism). Risk
factors for suicide can change with the individual case. Over time, they may
decrease or multiply.
Roberts et al. (1998) explored the risk of suicidal plans and ideation,
depression, and factors such as self-esteem, loneliness, and pessimism among
adolescents with a lifetime history of attempted suicide. Those with a lifetime
history of suicide attempts were more likely to think about death, wish to be
dead, think about suicide, and plan suicide. In examining the role of
depression in relation to suicidal thinking and attempts, these researchers
determined that depression clearly played a significant role, and in
combination with a history of previous attempts, the risk of recent ideation
was high. Additionally, it was determined that those who exhibited more
suicidal ideation were most likely to be lonely, fatalistic, and pessimistic
and to have lower self-esteem and higher levels of stress.
Boergers et al. (1998) explored psychological functioning and self-reported
reasons for suicide attempts in 120 adolescents. They determined that more than
half of the adolescents indicated that their attempts to die were to escape or
obtain relief. Those who indicated that the suicide attempts were in order to
die were significantly more depressed, hopeless, angry, and perfectionistic.
Depression in this study was found to be the most reliable predictor of the
motivation to die. Clearly, Mood Disorders put this population (e.g.,
adolescents) at risk for suicide. Assessing this risk potential and taking
steps to ensure the person’s safety is a top priority.
Adverse life events in combination with Depression and/or Substance-Related
Disorders may also lead to suicide. Other related factors may include: prior
suicide attempt, family history of mental disorder or substance abuse, firearms
in the residence, impulsive or aggressive tendencies or behaviors,
incarceration, and exposure to the suicidal behavior of others (e.g., family,
peers).
A “benchmark” finding on suicide has been established in persons where a
Mood Disorder and/or Substance Abuse Disorder is at work. Research has shown
that more than 90% of people who kill themselves have Depression and/or another
diagnosable mental or Substance Abuse Disorder, often in combination with other
mental disorders (Conwell et al., 1995; NIMH, 2003; Moscicki, 2001; Minino et
al., 2002).
CLOSING
REMARKS
Suicide completers can be divided into at least three subgroups according to
co-occurring disorders or comorbidity: a low comorbidity group, a
substance-dependent group, and a group exhibiting childhood onset of
psychopathology or mental disorders (Kim et al., 2003). James et. al. (2004)
reviewed the evidence of a possible association between AD/HD and suicide. The
electronic databases--Medline and Psy Lit--for 1966 through 2003 were searched
looking for articles on AD/HD and suicide. An association of AD/HD and
completed suicide was found, especially for young males. These researchers
concluded that AD/HD appears to increase the risk for suicide in males via the
“severity of comorbid disorders.” Identification and careful monitoring of
those at-risk, particularly males, may represent a clinically useful means of
reducing suicide in these cases.
Many interventions designed to reduce suicidality also include the treatment
of “mental” and “substance abuse” disorders. Preventive efforts to reduce
suicide should be based on research that shows which “risk factors” and
“protective factors” can be modified. Research continues to reinforce the
crucial variable of family members and close friends being involved in the
intervention and treatment process.
Just as people can die of heart disease or cancer, people can die from
mental illness. Suicide is almost always complicated, resulting from a
combination of painful suffering, desperate hopelessness, and underlying psychiatric
illness.
Is it possible to predict suicide? At the present time, there is no
definitive measure to predict suicide or suicidal behavior; very few persons
will actually commit suicide. However, we need to be fully aware that
researchers have identified risk factors pertinent to suicidal behavior. The
alarming numbers of suicide deaths and attempts emphasize the need for
carefully designed prevention programs. The “early” recognition,
identification, and treatment of the risk factors associated with suicide is
the single most important intervention for preventing and reducing suicidal
behavior.
REFERENCES
American Psychiatric Association. (2000). Diagnostic and statistical
manual of mental disorders (fourth edition, text revision). Washington, DC:
Author.
Barkley, R. A. (1997). AD/HD and the nature of self-control. New
York: Guilford Press.
Barkley, R. A. (1998). Attention deficit hyperactivity disorder: A
handbook for diagnosis and treatment. New York: Guilford Press.
Barkley, R. A. (2003). Attention deficit hyperactivity disorder. In E. J.
Mash & Barkley, R. A. (Eds.), Child psychopathology (pp. 75-143).
New York: Guilford Press.
Biederman, J. et. al. (1991). Comorbidity of attention deficit hyperactivity
disorder with conduct, depressive, anxiety, and other disorders. American
Journal of Psychiatry, 148, 564-577.
Biederman, J. et al. (1993). Patterns of psychiatric comorbidity, cognition,
and psychological functioning in adults with attention deficit hyperactivity
disorder. American Journal of Psychiatry, 150, 1792-1798.
Biederman, J. et al. (1995). Psychoactive substance use disorder in adults
with attention deficit hyperactivity disorder: Effects of ADHD and psychiatric
comorbidity. American Journal of Psychiatry, 152, 1652-1658.
Brown, T. E. (2000). Attention-deficit disorders and comorbidities in
children, adolescents, and adults. Washington, DC: American Psychiatric
Press. Journal of the American Academy of Child and Adolescent Psychiatry,
37, 1287-1293.
Boergers, J. et al. (1998). Reasons for adolescent suicide attempts:
Associations with psychological functioning. Journal of the American
Academy of Child and Adolescent Psychiatry, 37, 1287-1293.
Castellanos, X. F. et al. (1994). Quantitative morphology of the caudate
nucleus in Attention-Deficit/Hyperactivity Disorder. American Journal of
Psychiatry, 151, 1791-1796.
Castellanos, X. F. et al. (1996). Quantitative magnetic brain imaging in
Attention-Deficit/Hyperactivity Disorder. Archives of General Psychiatry,
53, 607-616.
Conwell, Y., & Brent, D. (1995). Suicide and aging I: Patterns of
psychiatric diagnosis. International Psychogeriatrics, 7, 149-164.
Department of Health and Human Services. (1997). Ninth special report to
the U. S. Congress on alcohol and health. Washington, DC: Author.
Eisner, A., & McClelland, J. (1999). Drugs of abuse. In J. S. Werry
& M. G. Aman (Eds.), Practitioner’s guide to psychoactive drugs for
children and adolescents (2 nd ed.). New York: Plenum Medical Books.
Erk, R. R. (2000). Five frameworks for increasing understanding and
effective treatment of attention-deficit/hyperactivity disorder: Predominantly
inattentive type. Journal of Counseling and Development, 78, 389-399.
Erk, R. R. (2004a). Attention-deficit/hyperactivity disorder in children and
adults. In R. R. Erk (Ed.), Counseling treatment for children and
adolescents with DSM-IV-TR disorders (pp. 109-154). Upper Saddle River,
NJ: Merrill Prentice Hall.
Erk, R. R. (2004b). Understanding the development of psychopathology in
children and adolescents. In R. R. Erk (Ed.), Counseling treatment for
children and adolescents with DSM-IV-TR disorders (pp. 37-87). Upper
Saddle River, NJ: Merrill Prentice Hall.
Field, L., & Seligman, L. (2004). Mood disorders in children and
adolescents. In R. R. Erk (Ed.), Counseling treatment for children and
adolescents with DSM-IV-TR disorders (pp. 240-273). Upper Saddle River,
NJ: Merrill Prentice Hall.
Geller, B., & Luby, J. (1997). Child and adolescent bipolar disorder: A
review of the past 10 years. Journal of American Academy of Child and
Adolescent Psychiatry, 36, 1186-1176.
Gredler, M. E. (2005). Learning and instruction: Theory into practice.
Upper Saddle River, NJ: Merrill Prentice Hall.
Harris, J. R. (1995). Where is the child’s environment? A group
socialization theory of development. Psychological Review, 102,
458-489.
Hechtman, L. (1992). Long-term outcome in attention-deficit hyperactivity
disorder. Psychiatric Clinics North America, 1, 553- 565.
James, A. et al. (2004). Attention deficit hyperactivity disorder and
suicide: A review of possible associations. Acta Psychiatrica Scandinavicia,
110, 408-412.
Johnson, L. D. et al. (1993). National survey results on drug use,
1975-1992 (NIH Publication 93-3597, Vol. I, Secondary School Students).
Rockville, MD: National Institute on Drug Abuse.
Kim, C. D. et al. (2003). Patterns of comorbidity in male suicide
completers. Psychological Medicine, 33, 1299-1309.
King, C. A. et al. (1996). Predictors of comorbid alcohol and substance
abuse in depressed adolescents. Journal of the American Academy of Child
and Adolescent Psychiatry, 35, 743-751.
Klassen, A. F. et al. (2004). Health-related quality of life in children and
adolescents who have a diagnosis of attention-deficit hyperactivity disorder. Pediatrics,
114, 541-547.
Laverstein, B. (1995). Neurological comorbidity patterns /differential
diagnosis in adult attention deficit disorder. In K. G. Nadeau (Ed.), A
comprehensive guide to attention deficit disoder in adults (pp. 74-108).
New York: Brunner/Mazel.
Lewinsohn, P. M. et al. (1991). Comorbidity of Unipolar Depression: I. Major
Depression with Dysthymia. Journal of Abnormal Psychology, 100,
205-213.
Lewinsohn, P. M. et al. (1995). Adolescent psychopathology: IV. Specificity
of psychosocial risk factors for depression and substance abuse in older
adolescents. Journal of the American Academy of Child and Adolescent
Psychiatry, 34, 1221-1229.
Lewinsohn, P. M. et al. (1998). Major depressive disorder in older
adolescents: Prevalence, risk factors, and clinical implications. Clinical
Psychological Review, 18, 765-794.
Mannuzza, S., K. et al. (1993). Adult outcome of hyperactive boys:
Educational achievement, occupational rank and psychiatric status. Archives
General Psychiatry, 50, 565-576.
Mann, J. J. et al. (1999). The neurobiology of suicide risk: A review for
the clinician. Journal of Clinical Psychiatry, 60 (Suppl 2), 7-11.
Minino, A. M., et al. (2002). Deaths: Final data for 2000. National
Vital Statistics Reports, 50 (15). Hyattsville, MD: National Center for
Health Statistics.
Mohr, D. C. (1995). Negative outcome in psychotherapy: A critical review. Clinical
Psychology: Science and Practice, 2, 1-27.
Moscicki, E. K. (2001). Epidemiology of completed and attempted suicide:
Toward a framework for prevention. Clinical Neuroscience Research,
310-323.
Nadeau, K. G. (1995). A comprehensive guide to attention deficit
disorder in adults: Research, diagnosis, and treatment. New York: Brunner/Mazel.
National Institute of Mental Health. (2003). Retrieved April 15, 2005, from http://www.nimh.nih.gove/publicat/harmaway.cfm
Piazza, N. (2004). Substance-related disorders. In R. R. Erk (Ed.), Counseling
treatment for children and adolescents with DSM-IV-TR disorders (pp.
273-303). Upper Saddle River, NJ: Merrill Prentice Hall.
Pliszka, S. A. et al. (1999). ADHD with comorbid disorders: Clinical
assessment and management. New York: Guilford Press.
Quay, H. C. (1997). Inhibition and attention deficit hyperactivity disorder.
Journal of Abnormal Child Psychology, 25, 7-13.
Quinn, P. O. (1995). Neurobiology of attention deficit disorder. In K. G.
Nadeau (Ed.), A comprehensive guide to attention deficit disorder in
adults: Research, diagnosis, and treatment (pp. 18-31). New York:
Brunner/Mazel.
Reinecke, M. A. (1995). Comorbidity of conduct disorder and depression among
adolescents: Implications for assessment and treatment. Cognitive and
Behavioral Practice, 2, 299-326.
Resnick, R. J. (2000). The hidden disorder: A clinicians guide to
attention deficit hyperactivity disorder in adults. American Washington,
DC: Psychological Association.
Roberts et al. (1998). Suicidal thinking among adolescents with a history of
attempted suicide. Journal of the American Academy of Child and Adolescent
Psychiatry, 37, 1294-1300.
Stowell, R. J. A., & Estroff, T. W. (1992). Psychiatric disorders in
substance-abusing adolescent inpatients: A pilot study. Journal of the
American Academy of Child and Adolescent Psychiatry, 31, 1036-1040.
Tarter, R. E., & Edwards, K. (1988). Psychological risk factors
associated with the risk for alcoholism. Clinical and Experimental Research,
12, 471-480.
Weiss, M., & Murray, C. (2003). Assessment and management of
attention-deficit hyperactivity disorder in adults. Canadian Medical
Association Journal, 18, 715-722.
Wilens, T. E., et al. (1996). Does ADHD affect the course of substance
abuse? Findings from a sample of adults with and without ADHD. American
Journal of Addiction, 7, 156-163,
Wilens, T. E. (1998). AOD use and attention-deficit/hyperactivity disorder. Alcohol
Health and Research World, 22. 127-131.
Wilens, T. E. et al. (1997). Attention deficit hyperactivity disorder (ADHD)
is associated with early onset substance use disorders. Journal of Nervous
and Mental Disease, 185, 475-482.
Wilens, T. E. Biederman, J., & Spencer, T. J. (2002). Attention deficit/hyperactivity
disorder across the lifespan. Annual Review of Medicine, 53, 113-232.
Werry, J. (1995, Summer). Q & A: An interview with John Werry, M.D. Attention!
The Magazine of Children and Adults with Attention Deficit Disorders, 2,
7-31.
Zetmetkin, A. J. (1986). The pathophysiology of attention deficit disorder
with hyperactivity: A review. In B. B. Lahey & A. E. Kazdin (Eds.), Advances
in clinical child psychology (pp. 177-216). New York: Plenum Press.
Zemetkin, A. J. (1990). Cerebral glucose metabolism in adults with
hyperactivity of childhood onset. New England Journal of Medicine,
323, 1361-1366.
Zemetkin, A. J. et al. (1996). Brain metabolism in teenagers with
attention-deficit hyperactivity disorder. Archives of General Psychiatry,
50, 333-340.
Disclaimer: The information contained on this web page is intended to be for
informational purposes only. It is not intended as psychological or medical
advice. Individuals who are diagnosed with AD/HD should secure information and
recommendations on AD/HD from their treating professional. A systems oriented
approach to AD/HD with families, schools, and communities working together and
multidimensional treatment for AD/HD are the focus of this web page and its
contents. The University of Tennessee at Martin is not responsible for the
information or views expressed herein. Last updated June, 2005. Copyright 1997.
All rights reserved. Email: rerk@utm.edu
INTERESTING LINKS FOR MORE INFORMATION ON AD/HD
· Children and Adults With Attention Deficit Disorders (CHADD)
· School Psychology Resources Online
· American Counseling Association
· American School Counselor Association
· American Mental Health Counselors Association
· American Psychological Association
· American Psychiatric Assocation
· Attention-Deficit Disorder Archives-Legal
· National Institute of Mental Health
This is the time this file has been accessed.
Email: rerk@utm.edu